By F. K. Mostofi (auth.), F. K. Mostofi M.D. (eds.)
During the prior decade there was an expanding know-how of the necessity for a unique method of the matter of bilharziasis. We do be aware of that 180-200 million individuals are contaminated and that the an infection is expanding yet we haven't as but been capable of resolution the query: Are they struggling with a ailment. examine of important data or clinic documents, mass biochemical or immunological checks and group surveys haven't but supplied the total solution. steadily and maybe begrudgingly, now we have come to achieve that we needs to learn the ma- first and foremost via good managed scientific observations to procure facts for the altered body structure, if any, because of bilharzial an infection; secondly and both very important via pathological experiences on person sufferers to figure out the structural adjustments, if any, due to an infection with this parasite; and through publish mortem reviews. Experimental reviews of bilharzial an infection in animals, beneficial as they are for the elucidation of the pathogenesis of the lesions within the laboratory animal and in guy can't function an alternative choice to the best details at the nature of the prospective lesions prompted via bilharziasis in guy. essentially, the importance of any scientific remark can 'be evaluated basically via trustworthy pathological confirmation.
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Extra resources for Bilharziasis: International Academy of Pathology · Special Monograph
2, 883-915 (1953). [32J MOUSA, A. , and A. GAREM: The haemodynamic study of Egyptian hepatosplenic bilharziasis. J. Egypt. Med. Ass. 42, 444-455 (1959). [33J ONSY, A. : The pathogenesis of endemic (Egyptian) splenomegaly. Trans. Roy. Soc. Trop. Med. Hyg. 30, 597-600 (1937). , H. SANDSTEAD, A. SCHULBERT, and A. EL ROUBY: Urinary excretion of zinc in patients with the syndrome of anaemia, hepatosplenomegaly, dwarfism and hypogonadism. J. Lab. Clin. Med. 62, 591-599 (1963). , A. ABDALLAH, A. EL ROOBY, M.
Blood examination reveals anemia (chlorosis), leucocytosis with a relatively high eosinophilia (50% or more). Such symptoms are due to toxemia [4J, and are followed 2 to 4 months later, the time taken by the worms in migrating to the lower urinary organs, by those of bladder invasion, namely, haematuria, dysuria and painful frequent micturation. 7. H aematuria Before the discovery of the worms, French authors and others had spoken of Egypt as the "Land of menstruating men"; and indeed, until recently, haematuria was considered by young bilharzial patients as a sign of virility.
27, 237-250 (1961). , A. NOMIER, and R. ZAHER: Evaluation of portal hypertension in hepatic schistosomiasis. Alexandria Med. J. 8, 208-219 (1962). [7J DAY, H. , and A. R. FERGUSON: An account of a form of splenomegaly with hepatic cirrhosis, endemic in Egypt. Ann. Trop. Med. Parasit. 3, 379-394 (1909). The etiology of Egyptian splenomegaly and hepatic cirrhosis. Trans. Roy. Soc. Trop. [8J Med. Hyg. 18, 121-130 (1924). [9J Bilharzial cirrhosis (Egyptian splenomegaly). J. Trop. Med. Hyg. 36, 17-·23 (1933).